Coconut Oil And Stomach Acid

By Dr. Sanford Pinna

The Helico Pylori Bacteria is one of the most common bacteria that infect the Human race. It is found in families, who pass the bacteria to each other. H. Pylori inhabits the stomach and the esophagus.It stimulates cells in the stomach to produce excessive amounts of gastric or hydrochloric acid.

This acid reflects back and up into the stomach causing “Heart burn” or, technically, Gastro-Esophogeal Reflux /Disease, commonly referred to as GERD.

This reflux is painful and can cause ulcers, gastritis and occasional gastric cancer, which can be deadly.

Coconut oil is an excellent “anti-biotic”. It kills bacteria and fungi on contact! Coconut oil is made of saturated fatty acids of the medium length variety. Its major Saturated Acid is called LAURIC ACID. The Lauric Acid invades the cell wall and destroys it.

TREATMENT

There are dozens of diagnostic methods for determining the presence of H. Pylori, and dozens of treatments devised by major pharmaceutical companies who make billions of dollars with disease.

The normal treatment consist of an accurate diagnosis using an analysis of breath from the patient which contains gas byproducts of the bacterium, followed by an average ten day treatment of six antibiotics daily, plus two proton pump inhibitors daily. (Prevacid, etc.) At the end, the patient is retested for the presence of H. Pylori.

There is an approximate 90 percent success rate, sometimes 70 to 80 percent.

DRAWBACKS OF MEDICAL TREATMENT

1. COST: $300 – $500, if no insurance.

2. SIDE EFFECTS: diarrhea, stomach complaints, overgrowth of bad bacteria.

3. TIME COSTS: visits to doctors and laboratories.

 

SUMMARY

Treating H. Pylori infections medically, is costly, time consuming, produces side effects and is not one hundred percent successful.

ALTERNATIVE TREATMENTS

Many of my patients who have studied alternative medicine on the Internet, have asked my opinion aboutVirgin Coconut Oil for the treatment of Pylori Infections causing hyperacidity.

HOW SHOULD I ADVISE?

My method of studying a new plant or drug is to first learn the chemistry of the plant or drug. I must know the molecular structure and how the molecule interacts with other molecules in the body.

Without this knowledge I would be like a carpenter that doesn’t know whether a nail will pierce steel. This knowledge requires research into the description of the molecules, its various chemical properties and thephysiological properties of the body molecules that will be affected.

Over thirty years as a Physician makes this task arduous, HOWEVER, I have learned to do the research and abstract the conclusion.

 

 

COCONUT OIL AND HOW IT KILLS BACTERIA

Coconut oil is an excellent “anti-biotic”. It kills bacteria and fungi on contact! Coconut oil is made of saturated fatty acids of the medium length variety. Its major Saturated Acid is called LAURIC ACID. The Lauric Acid invades the cell wall and destroys it.

Here is an excerpt from a scientific study:

“Studies on lipids in the 1960s by Kabara and colleagues showed medium-chain (C-8 to C-14) FAs and their monoglycerides to have antimicrobial effects against several laboratory organisms.

In the 1990s, more laboratory studies confirmed the antimicrobial activity of these lipids against gram-positive and some gram-negative organisms, including Neisseria gonorrhoeae, Helicobacter pylori, andChlamydia trachomatis, as well as Candida albicans yeast and enveloped viruses.

Since 1998, some clinical studies have confirmed these laboratory data, specifically data on monolaurin, the monoglyceride of lauric acid from VCO. A 2% gel preparation of Lauricidin (Skin Sciences Laboratory, Inc, Pasig City, Philippines), which contains 90% pure monolaurin, significantly degermed SA cultured from health workers’ hands after hospital duty.

Another study cultured the skin lesions of 100 pediatric patients. The top isolates were SA, coagulase-negative SA, Streptococcus spp, Enterobacter spp, and Escherichia vulneris. The sensitivity of these organisms to penicillin, oxacillin, erythromycin, fusidic acid, mupirocin, and vancomycin varied significantly, demonstrating low to high susceptibility, across the different isolates (Fisher exact test = 0.000; p < .05).

In marked contrast, sensitivity to monolaurin did not significantly differ across the different bacterial isolates (Fisher exact test = 0.110; p > .05), reflecting high antibacterial activity.

 

There also was a statistically significant and marked difference in resistance rates. SA, coagulase-negative SA, and Streptococcus spp did not exhibit any resistance to monolaurin as opposed to the varying resistance observed with the other antibiotics in this study.”

We can easily see that “MONOLAURIN” or LAURIC ACID, the most common fatty acid in coconut oil is “highly antibacterial” and kills “H. Pylori.”

 

Source:

http://drpinna.com/virgin-coconut-oil-and-stomach-acid-16443